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[PDF] Development of a micro flow-through cell for high field NMR spectroscopy.

[PDF] from sandia.govTM Alam… - 2011 - prod.sandia.gov
... 10 Figure 2: Photos of 1.5mm OD micro flow cell. a) unfilled flow cell. ... 12 Figure 3: Summary of
1H NMR data collected from 1.5 mm OD and 2.0 mm OD flow cells; a) comparison of data collected
from 1.5 mm OD cell: i. 600 MHz broadband probe, ii. 600 MHz diffusion probe, iii. ...
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FGL2/Fibroleukin Mediates Hepatic Reperfusion Injury by Induction of Sinusoidal Endothelial Cell and Hepatocyte Apoptosis in Mice

…, M Xue-Zhong, J Manuel, OD Rotstein… - Journal of …, 2011 - Elsevier
Hepatic ischemia and reperfusion was induced in wild type mice (WT) and in mice with deletion
or inhibition of FGL2 and FcRIIB. Liver injury was determined by AST release, necrosis and animal
death. Apoptosis was evaluated with caspase 3 and TUNEL staining. Results: FGL2 ...

CHEK2 genomic and proteomic analyses reveal genetic inactivation or endogenous activation across the 60 cell lines of the US National Cancer Institute

…, A Monks, RH Shoemaker, OD Abaan… - Oncogene, 2011 - nature.com
CHEK2 encodes a serine/threonine kinase (Chk2) activated by ATM in response to DNA
double-strand breaks. On the one hand, CHEK2 has been described as a tumor suppressor
with proapoptotic, cell-cycle checkpoint and mitotic functions. On the other hand, Chk2 is ...

PERP regulates enamel formation via effects on cell–cell adhesion and gene expression

[PDF] from ucsf.edu…, T Pramparo, LD Attardi, OD Klein - Journal of Cell …, 2011 - jcs.biologists.org
Little is known about the role of cell–cell adhesion in the development of mineralized
tissues. Here we report that PERP, a tetraspan membrane protein essential for epithelial
integrity, regulates enamel formation. PERP is necessary for proper cell attachment and ...
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Opposing Effects of Glutamine and Asparagine Govern Prion Formation by Intrinsically Disordered Proteins

…, R Krishnan, N Lyle, CW O'Donnell, OD King… - Molecular Cell, 2011 - Elsevier
Sequences rich in glutamine (Q) and asparagine (N) residues often fail to fold at the monomer
level. This, coupled to their unusual hydrogen-bonding abilities, provides the driving force to
switch between disordered monomers and amyloids. Such transitions govern processes ...
All 2 versions

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