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Interleukin-17A Promotes Early but Attenuates Established Disease in Crescentic Glomerulonephritis in Mice

D Odobasic, PY Gan, SA Summers… - The American Journal of …, 2011 - Elsevier
T helper (Th)17 cells might contribute to immune-mediated renal injury. Thus, we sought to define
the time course of IL-17A-induced kidney damage and examined the relation between Th17
and Th1 cells in a model of crescentic anti-glomerular basement membrane ...
All 2 versions

Addition of Angiotensin II Type 1 Receptor Blocker to CCR2 Antagonist Markedly Attenuates Crescentic Glomerulonephritis

[HTML] from nih.govM Urushihara, N Ohashi, K Miyata, R Satou… - …, 2011 - Am Heart Assoc
Kluwer Health, 351 West Camden Street, Baltimore, MD 21202-2436. Phone:
410-528-4050. Permissions: Permissions & Rights Desk, Lippincott Williams & Wilkins, a division
of Wolters ... Addition of Angiotensin II Type 1 Receptor Blocker to
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The IL-27 Receptor Has Biphasic Effects in Crescentic Glomerulonephritis Mediated Through Th1 Responses

SA Summers, RKS Phoon, JD Ooi… - The American Journal of …, 2011 - Elsevier
Despite its initially defined role as a T-helper type 1 cell (Th1)–inducing cytokine,
interleukin-27 (IL-27) has complex roles in vivo. The role of IL-27 receptor (IL-27R) was defined
in experimental crescentic glomerulonephritis induced by a foreign antigen, sheep ...
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… CD14++ CD16+ MONOCYTESENDOTHELIN RECEPTORS MARKEDLY AGGRAVATE CRESCENTIC RAPIDLY PROGRESSIVE GLOMERULONEPHRITIS IN …

D Mangieri, A Palmisano, I Libri, D Corradi… - NDT …, 2011 - ndtplus.oxfordjournals.org
INTRODUCTION AND AIMS: Idiopathic retroperitoneal fibrosis (IRF) is a rare immune-mediated
condition characterised by a fibro-inflammatory periaortic tissue which often entraps the ureters
and may cause acute renal failure. IRF is histologically characterized by marked fibrosis, ...

Role of amino acid transporter LAT2 in the activation of mTORC1 pathway and the pathogenesis of crescentic glomerulonephritis

R Kurayama, N Ito, Y Nishibori, D Fukuhara… - Laboratory …, 2011 - nature.com
Molecular mechanisms and signaling pathways leading to cellular proliferation and lesion formation
in the crescentic glomerulonephritis (CGN) remain elusive. In the present study we have explored
a potential role of the mammalian target of rapamycin complex 1 (mTORC1) signaling ...
Related articles - All 3 versions

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