L Han… - The American Journal of Pathology, 2011 - Elsevier Transforming growth factor (TGF)-β and fibroblast growth factor (FGF)-2 both promote repair in valve interstitial cell (VIC) injury models; however, the relationship between TGF-β and FGF-2 in wound repair are not well understood. VIC confluent monolayers were wounded ... Related articles - All 4 versions
P Thayer, K Balachandran, S Rathan… - Annals of biomedical …, 2011 - Springer 1W.H. Coulter School of Biomedical Engineering, Georgia Institute of Technology, Atlanta, GA, USA; 2School of Chemical and Biomolecular Engineering, Georgia Institute of Technology, Atlanta, GA, USA; and 3Department of Cardiology, Emory University, Atlanta, ... Cited by 4 - Related articles - All 4 versions
X Gu… - American Journal of Physiology-Heart …, 2011 - Am Physiological Soc The differentiation of valvular interstitial cells (VICs) to a myofibroblastic or osteoblast-like phenotype is commonly found in calcific valvular stenosis, although the molecular-level mechanisms of this process remain poorly understood. Due to the role of the Rho pathway in various ... Cited by 1 - Related articles - All 3 versions
KJ Rodriguez, LM Piechura… - Matrix Biology, 2011 - Elsevier Disruption of the extracellular matrix (ECM) is frequently found in calcific aortic valve disease (CAVD), yet the role of ECM components in valvular interstitial cell (VIC) function and dysfunction remains poorly understood. This study examines the contributions of exogenous and ... Cited by 2 - Related articles - All 2 versions
[CITATION] Persistent [beta]-Catenin Activation Underlies Senescence of Interstitial Cell of Cajal (ICC) Stem Cells in Naturally Aging Mice
DT Asuzu, Y Hayashi, MR Bardsley, G Farrugia… - Gastroenterology, 2011 - Elsevier
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