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[HTML] Lipid-induced insulin resistance mediated by the proinflammatory receptor TLR4 requires saturated fatty acid–induced ceramide biosynthesis in mice

[HTML] from nih.govWL Holland, BT Bikman, LP Wang… - The Journal of clinical …, 2011 - ncbi.nlm.nih.gov
1 Department of Internal Medicine, University of Texas Southwestern Medical Center,
Dallas, Texas, USA. 2 Program in Cardiovascular and Metabolic Diseases, Duke-National University
of Singapore Graduate Medical School, Singapore. 3 Sarah W. Stedman Nutrition and ...
Cited by 3 - Related articles - All 5 versions

Calpain-10 interacts with plasma saturated fatty acid concentrations to influence insulin resistance in individuals with the metabolic syndrome

P Perez-Martinez, J Delgado-Lista… - … American Journal of …, 2011 - Am Soc Nutrition
1 From the Lipid and Atherosclerosis Unit, IMIBIC/Reina Sofia University Hospital/University
of Cordoba, and CIBER Fisiopatologia Obesidad y Nutricion, Instituto de Salud Carlos III,
Córdoba, Spain (PP-M, JD-L, AG-R, and JL-M); the Nutrigenomics Research Group, ...
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Comment on the article “A saturated fatty acid–rich diet induces an obesity-linked proinflammatory gene expression profile in adipose tissue of subjects at risk of …

I Dahlman - The American journal of clinical nutrition, 2011 - Am Soc Nutrition
The effect of nutrient composition on human adipose gene expression, independently of body
mass index, is largely unknown. It was therefore with great interest that I read the article by van
Dijk et al (1) in a recent issue of the Journal. In their article, van Dijk et al report that ...
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Interruption of triacylglycerol synthesis in the endoplasmic reticulum is the initiating event for saturated fatty acid-induced lipotoxicity in liver cells.

MD Mantzaris, EV Tsianos… - The FEBS journal, 2011 - ncbi.nlm.nih.gov
The aim of the present study was to investigate in detail the molecular mechanisms by which
free fatty acids induce liver toxicity in liver cells. HepG2 and Huh7 human liver cell lines were
exposed to varying concentrations of stearate (18:0), oleate (18:1), or mixtures of the two ...
Cited by 1 - Related articles

Cdc42 and Rac1 are major contributors to the saturated fatty acid-stimulated JNK pathway in hepatocytes

M Sharma, F Urano… - Journal of Hepatology, 2011 - Elsevier
The current model for JNK activation by SFA involves endoplasmic reticulum (ER) stress, which
induces JNK activation through an inositol requiring enzyme 1 (IRE1αApoptosis Regulating Kinase
1 (ASK1) dependent mechanism. Here, we find that SFA-induced JNK activation is not ...
All 3 versions

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