[HTML] from cjb.netJG Barrera, KR Jones, JP Herman… - The Journal of …, 2011 - neuro.cjb.net Central administration of glucagon-like peptide-1 (GLP-1) causes a dose-dependent reduction in food intake, but the role of endogenous CNS GLP-1 in the regulation of energy balance remains unclear. Here, we tested the hypothesis that CNS GLP-1 activity is required for normal ... Related articles - All 3 versions
N Luo, G Marcelin, SM Liu, G Schwartz… - Endocrinology, 2011 - Endocrine Soc Neuropeptide Y (NPY) and agouti-related peptide (AGRP) can produce hyperphagia, reduce energy expenditure, and promote triglyceride deposition in adipose depots. As these two neuropeptides are coexpressed within the hypothalamic arcuate nucleus and mediate a major portion of ... Cited by 1 - Related articles - All 4 versions
T Wells, D Relkovic, H Furby, I Guschina… - 2011 - endocrine-abstracts.org Prader–Willi syndrome (PWS) is a neurodevelopmental disorder caused by a lack of paternal gene expression from 15q11–q13 and is characterised by failure to thrive in infancy, followed by hyperphagia due to abnormal satiety responses and increased motivation by food. We ... Cached
ML Trujillo, C Spuch, E Carro… - Endocrinology, 2011 - Endocrine Soc The purpose of this work was to study the central mechanisms involved in food intake regulation and leptin resistance during gestation in the rat. Sprague Dawley rats of 7, 13, and 18 d of pregnancy [days of gestation (G) 7, G13, and G18] were used and compared with nonpregnant ... Cited by 1 - Related articles - All 6 versions
[CITATION]Hyperphagia and obesity in mice with genetic ablation of gastrointestinal brain-derived neurotrophic factor
EA Fox, JE Biddinger, KR Jones, J McAdams… - Autonomic …, 2011 - Elsevier
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