K Tsunoda, T Ota, M Saito, T Hata… - The American Journal of …, 2011 - Elsevier Pemphigus vulgaris is an autoimmune disease caused by IgG antibodies against desmoglein 3 (Dsg3). Previously, we isolated a pathogenic mAb against Dsg3, AK23 IgG, which induces a pemphigus vulgaris-like phenotype characterized by blister formation. In the present ... All 2 versions
H Takahashi, M Kouno, K Nagao, N Wada… - J Clin …, 2011 - Am Soc Clin Investig 1 Department of Dermatology, Keio University School of Medicine, Shinjuku-ku, Tokyo, Japan. 2 Center for Experimental Medicine and Systems Biology, Institute of Medical Science, University of Tokyo, Minato-ku, Tokyo, Japan. 3 Department of Microbiology and ...
ZG Compson, KC Larson, MS Zinkgraf… - Oecologia, 2011 - Springer Abstract We examined how the galling aphid Pemphigus batae manipulates resource translocation patterns of resis- tant and susceptible narrowleaf cottonwood Populus angustifolia. Using carbon-14 (14C)-labeling experiments in common garden trials, five patterns emerged. ... Related articles - All 2 versions
D Rafei, R Muller, N Ishii, M Llamazares… - The American Journal of …, 2011 - Elsevier Pemphigus is considered an autoimmune bullous skin disorder associated with IgG against the desmosomal components, desmoglein 3 (Dsg3) and desmoglein 1 (Dsg1). This concept is supported by the in vitro and in vivo pathogenicity of anti-Dsg3/Dsg1 IgG and the mucosal blistering ... Related articles - All 4 versions
HY Schultz, LA Diaz, DA Sirois, VP Werth… - Journal of Investigative …, 2011 - nature.com The third international meeting cosponsored by the International Pemphigus and Pemphigoid Foundation (IPPF) and the National Institute of Arthritis and Musculoskeletal and Skin Diseases was attended by approximately 130 scientists and clinicians representing the ... All 3 versions