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… T Cells Expressing IL-2 Receptor {alpha}-Chains (CD25). Breakdown of a Single Mechanism of Self-Tolerance Causes Various Autoimmune Diseases. J. Immunol. …

S Sakaguchi, N Sakaguchi, M Asano… - Journal of immunology …, 2011 - ncbi.nlm.nih.gov
... Pillars Article: Immunologic Self-Tolerance Maintained by Activated T Cells Expressing IL-2
Receptor {alpha}-Chains (CD25). Breakdown of a Single Mechanism of Self-Tolerance Causes
Various Autoimmune Diseases. J. Immunol. 1995. 155: 1151-1164. ...
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ROR [gamma] t drives production of the cytokine GM-CSF in helper T cells, which is essential for the effector phase of autoimmune neuroinflammation

L Codarri, G Gyülvészi, V Tosevski, L Hesske… - Nature …, 2011 - nature.com
Although the role of the T H 1 and T H 17 subsets of helper T cells as disease mediators in autoimmune
neuroinflammation remains a subject of some debate, none of their signature cytokines are essential
for disease development. Here we report that interleukin 23 (IL-23) and the transcription ...
Cited by 5 - All 3 versions

T helper 17 cell heterogeneity and pathogenicity in autoimmune disease

K Ghoreschi, A Laurence, XP Yang, K Hirahara… - Trends in …, 2011 - Elsevier
T helper (Th)17 cells have been proposed to represent a new CD4 + T cell lineage that is important
for host defense against fungi and extracellular bacteria, and the development of autoimmune
diseases. Precisely how these cells arise has been the subject of some debate, with ...

EMMPRIN: a novel regulator of leukocyte transmigration into the CNS in multiple sclerosis and experimental autoimmune encephalomyelitis

[HTML] from jneurosci.orgSM Agrawal, C Silva… - The Journal of …, 2011 - Soc Neuroscience
Extracellular matrix metalloproteinase inducer (EMMPRIN, CD147) is a member of the Ig
superfamily, with various physiological roles including the induction of matrix metalloproteinases
(MMPs), leukocyte activation, and tumor progression. In this study, we illustrate a novel ...
Cited by 3 - Related articles - All 2 versions

[HTML] Onset of autoimmune lymphoproliferative syndrome (ALPS) in humans as a consequence of genetic defect accumulation

[HTML] from nih.govA Magerus-Chatinet, B Neven… - The Journal of …, 2011 - ncbi.nlm.nih.gov
1 INSERM U768, Université Paris Descartes, Hôpital Necker-Enfants Malades, Paris,
France. 2 Unité d'Immunologie et d'Hématologie Pédiatrique, Assistance Publique des Hôpitaux
de Paris, Paris, France. 3 Royal Manchester Children's Hospital, University of ...
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