S Buschke, HJ Stark, A Cerezo… - Molecular Biology of …, 2011 - Am Soc Cell Biol The mechanism by which TGFβ regulates differentiation in human epidermal keratinocytes is still poorly understood. To assess the role of Smad signaling, we engineered human HaCaT keratinocytes either expressing siRNA against Smads2, 3, 4 or overexpressing Smad7 ... All 2 versions
[HTML] from molbiolcell.orgS Buschke, HJ Stark, A Cerezo… - Molecular Biology of …, 2011 - Am Soc Cell Biol The mechanism by which transforming growth factor-β (TGFβ) regulates differentiation in human epidermal keratinocytes is still poorly understood. To assess the role of Smad signaling, we engineered human HaCaT keratinocytes either expressing small interfering RNA against Smads2, 3, ... Related articles - All 2 versions
[HTML] from nih.govFPW Radner, S Grond, G Haemmerle… - Dermato- …, 2011 - ncbi.nlm.nih.gov Keratinocyte differentiation is essential for skin development and the formation of the skin permeability barrier. This process involves an orchestrated remodeling of lipids. The cleavage of precursor lipids from lamellar bodies by β-glucocerebrosidase, sphingomyelinase, phospholipases ... Related articles - All 6 versions
R Sestito, S Madonna, C Scarponi, F Cianfarani… - The FASEB Journal, 2011 - FASEB IL-22 has a pathogenetic role in psoriasis, where it is responsible for the altered proliferation and differentiation of keratinocytes and induces inflammatory molecules. The IL-22-induced effects are mediated by STAT3, whose activity is proportional to acetylation in lysine (Lys)685 ... Related articles - All 3 versions
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