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JC Jennette, H Xiao, R Falk… - Contrib Nephrol, 2011 - content.karger.com
Abstract Antineutrophil cytoplasmic autoantibodies (ANCA) are closely associated with systemic
small vessel vasculitis characterized by segmental vessel wall necrotizing inflam- mation and
a paucity of immunoglobulin deposition. Clinically, in vitro and experimental animal ...
Cited by 1 - Related articles - All 3 versions
J Bontscho, A Schreiber, RA Manz… - Journal of the …, 2011 - Am Soc Nephrol
Anti-neutrophil cytoplasmic autoantibodies (ANCA) cause vasculitis and necrotizing crescentic
glomerulonephritis (NCGN). Steroids and cytotoxic drugs reduce mortality but can cause significant
adverse events. The proteasome inhibitor bortezomib (BTZ) prevents glomerulonephritis ...
All 5 versions
G Moroni, C Casati, S Quaglini, B Gallelli… - …, 2011 - journals.lww.com
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Related articles - All 4 versions
D Odobasic, PY Gan, SA Summers… - The American Journal of …, 2011 - Elsevier
T helper (Th)17 cells might contribute to immune-mediated renal injury. Thus, we sought to define
the time course of IL-17A-induced kidney damage and examined the relation between Th17
and Th1 cells in a model of crescentic anti-glomerular basement membrane ...
All 2 versions
S Guo, TA Wietecha, KL Hudkins, Y Kida… - Kidney …, 2011 - nature.com
Mice transgenic for thymic stromal lymphopoietin (TSLP), under regulation of the
lymphocyte-specific promoter Lck, develop cryoglobulinemia and membranoproliferative glomerulonephritis
(MPGN) similar to the disease in patients. To determine whether infiltrating macrophages, ...

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