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[CITATION] Biological Mechanisms of Focal Glomerulosclerosis: A Major Cause of Acquired and Inherited Renal Disease

M Pollak - Annual Review of Medicine, 2011 - … Reviews 4139 El Camino Way, PO …
HJ Kim, T Sato, B Rodríguez-Iturbe… - Journal of Pharmacology …, 2011 - ASPET
The Imai rat is a model of spontaneous focal glomerulosclerosis, which leads to heavy
proteinuria, hyperlipidemia, hypertension, and progressive renal failure. Treatment with AT1
blockers (ARBs) ameliorates proteinuria, hyperlipidemia, and nephropathy in this model. ...
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C Wei, S El Hindi, J Li, A Fornoni, N Goes… - Nature Medicine, 2011 - nature.com
Focal segmental glomerulosclerosis (FSGS) is a cause of proteinuric kidney disease, compromising
both native and transplanted kidneys. Treatment is limited because of a complex
pathogenesis, including unknown serum factors. Here we report that serum soluble ...
Cited by 1
B Smeets, C Kuppe, EM Sicking… - Journal of the …, 2011 - Am Soc Nephrol
The pathogenesis of the development of sclerotic lesions in focal segmental glomerulosclerosis
(FSGS) remains unknown. Here, we selectively tagged podocytes or parietal epithelial cells
(PECs) to determine whether PECs contribute to sclerosis. In three distinct models of ...
All 3 versions
O Boyer, G Benoit, O Gribouval, F Nevo… - Journal of the …, 2011 - Am Soc Nephrol
The recent identification of mutations in the INF2 gene, which encodes a member of the formin
family of actin-regulating proteins, in cases of familial FSGS supports the importance of an intact
actin cytoskeleton in podocyte function. To determine better the prevalence of INF2 ...
Cited by 2 - Related articles - All 4 versions

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