K Dahlke, CD Wrann, O Sommerfeld… - The Journal of …, 2011 - Am Assoc Immnol Complement activation represents a crucial innate defense mechanism to invading microorganisms, but there is an eminent lack of understanding of the separate contribution of the different complement activation pathways to the host response during sepsis. We ... Related articles - All 3 versions
MD Burkitt, A Varro, JH Caamano… - Gut, 2011 - gut.bmj.com Results H. felis infection of wild-type mice resulted in gastric atrophy (29% fewer parietal cells were observed in infected than in control mice (p < 0.05, 1-way ANOVA and Holm Sidak post hoc test)), a 1.5-fold increase in the number of Ki67 positive proliferating cells and no ... All 2 versions
LA Tan, AC Yang, U Kishore… - Protein & Cell, 2011 - Springer 1 Medical Research Council Immunochemistry Unit, Department of Biochemistry, University of Oxford, South Parks Road, Oxford OX1 3QU, UK 2 Present address: Department of Immunology, Faculty of Medicine, Imperial College London, Hammersmith Hospital, Du ... Related articles - All 3 versions
U Kishore… - Immunobiology, 2011 - Elsevier C1q, the first subcomponent of the classical pathway, is a charge pattern recognition molecule that binds a diverse range of self, non-self and altered self ligands, leading to pro-inflammatory complement activation. Although complement is required for tissue homeostasis as well ...
[HTML] from biochemj.orgL Di-xian, CH Mei, M Jun, G Zachary… - Biochemical …, 2011 - biochemj.org AKR1B10 (aldo-keto reductase family 1, member B10) protein is primarily expressed in normal human small intestine and colon, but overexpressed in several types of human cancers and considered as a tumour marker. In the present study, we found that AKR1B10 protein is ... Cached
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