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K Dahlke, CD Wrann, O Sommerfeld… - The Journal of …, 2011 - Am Assoc Immnol
Complement activation represents a crucial innate defense mechanism to invading
microorganisms, but there is an eminent lack of understanding of the separate contribution of
the different complement activation pathways to the host response during sepsis. We ...
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MD Burkitt, A Varro, JH Caamano… - Gut, 2011 - gut.bmj.com
Results H. felis infection of wild-type mice resulted in gastric atrophy (29% fewer parietal cells
were observed in infected than in control mice (p < 0.05, 1-way ANOVA and Holm Sidak post
hoc test)), a 1.5-fold increase in the number of Ki67 positive proliferating cells and no ...
All 2 versions
LA Tan, AC Yang, U Kishore… - Protein & Cell, 2011 - Springer
1 Medical Research Council Immunochemistry Unit, Department of Biochemistry, University of
Oxford, South Parks Road, Oxford OX1 3QU, UK 2 Present address: Department of
Immunology, Faculty of Medicine, Imperial College London, Hammersmith Hospital, Du ...
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U Kishore… - Immunobiology, 2011 - Elsevier
C1q, the first subcomponent of the classical pathway, is a charge pattern recognition molecule
that binds a diverse range of self, non-self and altered self ligands, leading to pro-inflammatory
complement activation. Although complement is required for tissue homeostasis as well ...
[HTML] from biochemj.orgL Di-xian, CH Mei, M Jun, G Zachary… - Biochemical …, 2011 - biochemj.org
AKR1B10 (aldo-keto reductase family 1, member B10) protein is primarily expressed in normal
human small intestine and colon, but overexpressed in several types of human cancers and
considered as a tumour marker. In the present study, we found that AKR1B10 protein is ...
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